
Associate Professor
Psychiatry
Anna Molofsky MD, PhD studies the role of glial cells, particularly astrocytes, in neural circuit development and in psychiatric diseases.
Publications
Publisher Correction: Circuit and molecular architecture of a ventral hippocampal network.
Nature neuroscience
In situ and transcriptomic identification of microglia in synapse-rich regions of the developing zebrafish brain.
Nature communications
Reactive astrocyte nomenclature, definitions, and future directions.
Nature neuroscience
Circuit and molecular architecture of a ventral hippocampal network.
Nature neuroscience
Location, Location, Location: Transcriptional Control of Astrocyte Heterogeneity.
Trends in immunology
Astrocytes and Microglia: In Sickness and in Health.
Trends in neurosciences
The immune system and psychiatric disease: a basic science perspective.
Clinical and experimental immunology
Variation among intact tissue samples reveals the core transcriptional features of human CNS cell classes.
Nature neuroscience
Astrocyte-derived interleukin-33 promotes microglial synapse engulfment and neural circuit development.
Science (New York, N.Y.)
Dynamism of an Astrocyte In Vivo: Perspectives on Identity and Function.
Annual review of physiology
Chapter 36 Mechanisms of Astrocyte Development.
Patterning and Cell Type Specification in the Developing CNS and PNS
Regional astrocyte allocation regulates CNS synaptogenesis and repair.
Science (New York, N.Y.)
Regulated temporal-spatial astrocyte precursor cell proliferation involves BRAF signalling in mammalian spinal cord.
Development (Cambridge, England)
Astrocytes and disease: a neurodevelopmental perspective.
Genes & development
Bmi-1 over-expression in neural stem/progenitor cells increases proliferation and neurogenesis in culture but has little effect on these functions in vivo.
Developmental biology
Increasing p16INK4a expression decreases forebrain progenitors and neurogenesis during ageing.
Nature
Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.
Genes & development
Stem cell self-renewal and cancer cell proliferation are regulated by common networks that balance the activation of proto-oncogenes and tumor suppressors.
Cold Spring Harbor symposia on quantitative biology
Diverse mechanisms regulate stem cell self-renewal.
Current opinion in cell biology